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عمومی::
پاتوفیزیولوژی، پاتوفیزیولوژی، پاتوفیزیولوژی
This release may be suffi- cient to induce acute respiratory distress syndrome (ARDS) and systemic inflammatory response syndrome (SIRS).4,17 Conversely, ARDS can also trigger cerebral inflammation by releasing inflammatory mediators like cytokines.4,17 The ventilatory treatment for ARDS may create a therapeu- tic dilemma in the presence of cerebral pathophysiology.
Pathophysiology
This is most likely related to the complex pathophysiology of brain dam- age, the heterogeneous patterns of damage, and various preexisting patient diseases.
Hyponatremia is observed in 30% to 40% of patients with SAH, and is most commonly the result of cerebral salt-wasting syndrome (CSWS) or the syn- drome of inappropriate secretion of antidiuretic hormone (SIADH).193 Because these two conditions have different pathophysiology, correct diagnosis and management of hyponatremia is important, particularly when it precedes development of DCI and can worsen clinical outcome.
Orthostatic hypotension fol- lowing spinal cord injury: understanding clinical pathophysiology.،Ventricular Fibrillation Pathophysiology * Ventricles consist of areas of normal myocardium alternating with areas of ischemic, injured, or infarcted myocardium, leading to a chaotic asynchronous pattern of ventricular depolarization and repolarization.
Pulseless Electrical Activity Pathophysiology * Cardiac conduction impulses occur in an organized pattern but do not produce myocardial contraction (this condition was formerly called electromechanical dissociation), insufficient ventricular filling during diastole, or ineffective contractions.
Sinus Tachycardia (Figure 18) Pathophysiology * More of a physical response or sign than an actual arrhythmia or pathologic condition
Atrial Fibrillation (Figure 19) and Atrial Flutter (Figure 20) Pathophysiology * Atrial impulses are faster than sinoatrial (SA node) impulses.
May include AV nodal reentrant tachycardia or AV reentry tachycardia Pathophysiology * Reentry phenomenon: Impulses recycle repeatedly in the AV node because an abnormal rhythm circuit allows a wave of depolarization to travel in a circle.،This release may be suffi- cient to induce acute respiratory distress syndrome (ARDS) and systemic inflammatory response syndrome (SIRS).4,17 Conversely, ARDS can also trigger cerebral inflammation by releasing inflammatory mediators like cytokines.4,17 The ventilatory treatment for ARDS may create a therapeu- tic dilemma in the presence of cerebral pathophysiology.
Pathophysiology
This is most likely related to the complex pathophysiology of brain dam- age, the heterogeneous patterns of damage, and various preexisting patient diseases.
Hyponatremia is observed in 30% to 40% of patients with SAH, and is most commonly the result of cerebral salt-wasting syndrome (CSWS) or the syn- drome of inappropriate secretion of antidiuretic hormone (SIADH).193 Because these two conditions have different pathophysiology, correct diagnosis and management of hyponatremia is important, particularly when it precedes development of DCI and can worsen clinical outcome.
Orthostatic hypotension fol- lowing spinal cord injury: understanding clinical pathophysiology.
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