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عمومی::
پیش التهابی
Hyperbaric oxygen inhibits stimulus-induced proinflammatory cytokine synthesis by human blood-derived monocyte-macrophages.
Proinflammatory clearance of apoptotic neutrophils induces an IL-12(low)IL-10(high) regulatory phenotype in macrophages.
An unrestrained proinflammatory M1 macrophage population induced by iron impairs wound healing in humans and mice.
NOTCH reprograms mitochondrial metabolism for proinflammatory macrophage activation.
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