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عمومی::
همایندها
Identification of postnatal factors that affect the risk for and the course of retinopathy of prematurity might allow neonatologists and ophthalmologists to attempt to prevent the disease and to limit comorbidities with which it shares modifiable risk factors.
Comorbidities
It often occurs in conjunction with other neonatal morbidities such as neurological dysfunction, poor brain growth, necrotising enterocolitis, intraventricular haemorrhage, and bronchopulmonary dysplasia.113 In extremely preterm infants, severe retinopathy of prematurity predicts risk of death or major disability at age 11 years.114 Therefore addressing poor postnatal growth, hyperoxia, and infection and inflammation to reduce risk of retinopathy of prematurity might also reduce the risk of these comorbidities.
Ablative treatment of non-vascularised retina when the risk of retinal detachment is substantial helps to prevent blindness, but does not address the underlying cause of retinopathy of prematurity or other comorbidities, which is the failure of normal neural and vascular growth.
Prevention by reduction of risk factors that disrupt normal retinal vascularisation is likely to be more effective than late treatment of neovascularisation, not only with respect to vision, but also other comorbidities of premature birth.
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